Reduced cortical VPS26B levels are associated with altered glutamate receptor expression and synaptic protein loss in the primary motor cortex of a Parkinsonian mouse model
本研究证实,帕金森病小鼠模型初级运动皮层中VPS26B水平降低会导致表面GluA1减少和突触蛋白丢失,从而引起运动缺陷,而VPS26B过表达可部分挽救这些缺陷。