The inflammatory paradox of TLR4/NF-κB signaling in canine transmissible venereal tumors: why a protumoral pathway is associated with tumor stabilization
本综述提出,TLR4/NF-κB通路通过重编程炎症信号以抑制肿瘤生存机制并招募效应免疫细胞,从而矛盾地促进了犬传染性性传播肿瘤的稳定而非进展,同时强调了关键的研究空白并提出了验证该机制的实验策略。