Integrated Multi-Omics Analysis Identifies ATAD5 as a Mediator of Tumor Plasticity and Immunosuppressive Microenvironmental Remodeling in Castration-Resistant Prostate Cancer
本研究通过整合多组学分析与功能验证,确定了 ATAD5 是去势抵抗性前列腺癌中肿瘤可塑性与免疫抑制微环境重构的关键介质,并将其高表达与侵袭性临床结局及免疫浸润减少联系起来。