Neuroprotective effect of curcumin against reserpine-induced oxidative damage in the cerebellum of mice in an acute model of Parkinson's disease: Combined in vivo study, molecular docking and molecular dynamics
本研究表明,姜黄素通过减轻小脑氧化应激和炎症,在雷蛇精诱导的帕金森病小鼠模型中,对雷蛇精引起的氧化损伤和运动功能障碍发挥神经保护作用,分子对接与动力学模拟进一步证实了这一机制,即姜黄素与儿茶酚-O-甲基转移酶的结合相互作用比美多扶更强。